Preliminary studies suggest conolidine might inhibit specific ion channels, minimizing neuronal excitability and restricting discomfort signals. This mechanism is especially appropriate in neuropathic discomfort, where abnormal signaling results in persistent pain. In addition, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to ache notion. Computation... https://rodneyd936rpm0.mycoolwiki.com/user